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Targeting the elevated IFN- in vitiligo patients by human anti- IFN- monoclonal antibody hampers direct cytotoxicity in melanocyte
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But the mechanism is receptor-mediated and tissue-specific, meaning KPV produces robust effects in conditions where MC1R signaling drives inflammation and produces minimal effects where other pathways dominate
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Semple et al., 2010) indicated that within 24 h of the trauma, polymorphonuclear leukocytes and cytokines released inflammatory agents like IL-6, IL-1, and TNF-
JL: Writing original draft
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