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IGF-1 DES binds IGFBP-3 poorly, similar to LR3, but it also has roughly ten-fold higher potency at IGF-1R because the N-terminal tripeptide normally dampens receptor-binding affinity

Human studies provide topical and wound-care context [1] [2] [3] [4] [5] , while an animal wound model provides local-administration context [6]

Some members see low copays for medications, while others face higher out-of-pocket costs

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