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Additionally, regulatory approvals for drugs such as semaglutide (Wegovy) specifically for obesity treatment have expanded the patient population significantly
The question became engineering: could you build a molecule that activated glucagon enough to capture the fat-oxidation benefit without activating it so much that you re-introduced the glycemic penalty
While not every patient develops deficiencies, semaglutide slows nutrient absorption enough that most benefit from targeted supplementation
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