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While no specific studies have examined antibiotics as a trigger, many patients with ME/CFS have frequent infections treated with antibiotics [47, 69,70,71,72]
In neuronal senescence model studies, the active ingredient of the product can cross the blood-brain barrier (and act directly on neurons in vitro), increase NAD+ levels in nerve cells, protect mitochondrial function, reduce neuronal apoptosis, and promote the synthesis and release of neurotransmitters (such as dopamine and acetylcholine), thereby improving signal transduction in nerve cells
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