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However, sustained opening of the mPTP can result in membrane depolarization, reduced ATP production, and ultimately, mitochondrial dysfunction

However, when it is in excess, these benefits are overcome by a pro-inflammatory activity, being associated with worse haemodynamic and metabolic profile in patients affected by HFpEF [35, 36]

Death from myocardial infarction (MI) and stroke is 2- 4 times greater than death from coronary artery disease (CAD) in non-diabetic patients