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Conversely, brain IR, less recognized but equally important, pertains to impaired insulin signaling in neuronal and glial cells [6]
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Johnson BA
Inositol and semaglutide work through different metabolic pathways, so they do not directly interact pharmacologically
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The inhibition, which shows dose dependency, is exerted via interactions with receptors in the brain that are accessible via leaks in the blood-brain barrier of the circumventricular organs, mainly the postrema area, the subfornical organ and the median eminence [10, 11]
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