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The authors propound that deficient islet GABA signaling/content may contribute to the hyper-glucagonemia of T2D which again reinforces a role for GABA-based therapeutics

19 Clinical data thus far has been encouraging

PubMed 96

Most importantly, they are not to be construed as medical advice or therapeutic endorsement

Retatrutide adds glucagon receptor agonism on top of both, which is why the obesity field treats it as a step change rather than an incremental dose tweak