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They used reducible disulfide linkage to develop redox-sensitive mechanisms to enable intracellular MK-801 release, allowing for the summative cellular activities of GLP-1 agonists and NMDA antagonists
6a, b), as we observed in previous experiments with 3-PLG-A-GSH [24], and with serum albumin cargo loaded A-GSH targeted vesicular nanoparticles [32, 33]
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