For example, genetic deletion of GLP-1R results in an impaired left ventricular contractile response and diastolic function in mice.28 The stimulation of GLP-1R in cardiomyocytes has also been shown to increase their viability through activation of the RISK pathway (Fig
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doi:10.21037/tau.2016.11.16 Araujo AB, O'Donnell AB, Brambilla DJ, et al