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As discussed above, lipopolysaccharides (LPS) could prompt inflammation and amyloidogenesis in the brain, by inducing the disorder of TNF-a, IL-1, and cyclooxygenase-2 (COX-2) (87)

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This cycling approach allows the body to benefit from the peptide repair signaling during the active phase and then consolidate those gains during the off phase using its own endogenous repair mechanisms

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Efficacy and safety of coenzyme Q10 in heart failure: a meta-analysis of randomized controlled trials

Although weight loss likely plays a major role, emerging evidence suggests that anti-inflammatory and neuromodulatory mechanisms may also contribute