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doi:10.1097/AJP.0b013e318255655d

The GPR30 receptor agonist G1 inhibits ferroptosis and significantly improves chondrocyte viability and motor ability in mice through the YAP1/FTH1 pathway, while GPR30 receptor antagonist G15 has the opposite effect, suggesting that hormone-related ferroptosis may emerge as a promising target for alleviating osteoarthritis in postmenopausal women

PMID: 65

This edition explores whats real, whats hype, and how we might eventually personalize GHK-Cu through omics data

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