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Due to the high expression profile of GLP-1R in pancreatic -cells, GLP-1 analogs (such as exendin-4) have been modified by different methods (to prolong half-life, maintain receptor binding affinity and reduce renal drug accumulation) for theranostic purposes and serve as excellent templates for future cardiovascular drug development[178-184]
It works primarily through AMPK activation and multiple metabolic pathways, not through direct GLP-1 receptor binding
Furthermore, increasing studies have proven that GPX4 is the core regulator of ferroptosis (8)
They will guide you on dose adjustments, monitor any side effects, and help manage potential risks associated with treatment
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