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Similarly, GLP-1R agonists are candidate therapies for AD due to their role in modulating neuroinflammation and -amyloid accumulation

Exenatide activated eNOS and NO production in endothelial cells, induced dose-dependent vasorelaxation, and mitigated high-glucose or lipid-induced endothelial dysfunction in arterioles ex vivo, effects which were reduced by AMPK (AMP-activated protein kinase) inhibition [86]

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Despite their benefits, both medications come with potential side effects

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