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Deacetylation As the principal mitochondrial nicotinamide adenine dinucleotide (NAD+)-dependent deacetylase, Sirtuin3 regulates protein activity and stability through substrate-specific lysine deacetylation (Kac), thereby participating in pathophysiological processes such as cellular oxidative stress, autophagy, and cell death [36]

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Tranexamic acid and glutathione , each at a precise concentration of 1000 ppm, act as a powerful duo against hyperpigmentation