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These conditions share common mechanisms involving disrupted redox homeostasis, elevated oxidative stress, and impaired detoxification, underscoring the therapeutic potential of strategies aimed at restoring glutathione balance to mitigate disease progression

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RD16/0017/0012)

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They are formed in the body from normal metabolism (such as converting food to energy) but are increased by exposure to environmental toxins, such as cigarette smoke and air pollution

These immunobiotics have also been shown to reduce plasma T H 17 levels and inflammatory markers (Kanmani et al., 2024)