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Subcellular GSH levels Liver mitochondrial and cytosolic fractions were prepared from frozen liver samples as described 17

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Background and Summary of Retrospective Studies Homocystinuria caused by congenital CBS and MTHFR defects or by genetic alteration of cobalamin metabolism is characterized by clinical problems including frequent arterial and venous thromboembolism and premature atherosclerosis (McCully, 1983

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