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The research highlights that nanoplastics deplete the neural stem cell pool in the brain by impairing mitochondrial function and inducing cellular senescence, ultimately affecting neurogenesis in the hippocampal region and related cognitive functions, suggesting a close association between microplastics and the development of Alzheimers disease (39)
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The mitochondrial toxins rotenone and 1-methy-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP), two PD-associated inhibitors of mitochondrial complex I, can induce PD-like phenotypes both in vitro and in vivo [13]
In controlled environments, Epitalon is explored for its ability to interact with: Telomerase activity related to cellular lifespan Oxidative stress response and cellular balance Circadian rhythm and sleepwake cycle regulation Cellular repair and regeneration pathways This makes it particularly relevant in research models studying biological ageing and system regulation
Oxidative stress markers were assessed
