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This is then followed by the binding of arrestin, which sterically hinders coupling to G proteins, effectively desensitizing the receptor (Luttrell and Getsy-Palmer, 2010)

Significance for all tests was assumed for p 0.05 vs

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Tofacitinib is the first of a new class of oral drugs to have synthetic small molecules that interfere with specific signal-transduction pathway and is the third class of DMARD (tsDMARDs) in RA treatment

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