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This convergence of genetic and pharmacological perturbations supports a model in which ADORA2b ablation shifts RBC metabolism toward the PPP, while caffeine imposes a dual inhibitory effect blocking both ADORA2b signaling and G6PD enzymatic activity ultimately compounding the impairment of RBC energy and redox homeostasis
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Contains three amino acids: glutamate, glycine, and cysteine
19 Dysregulated putrescine metabolism may enhance MAOB activity, increasing GABA synthesis and exacerbating tonic inhibition under pathological conditions
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RO exhibit potent oxidative activity, abstracting hydrogen atoms and reacting with biomolecules, thereby causing membrane damage and functional impairment, as well as modulating protein functionality and genomic stability [67]
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