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When phosphorylated, AMPK promotes catabolic pathways such as fatty acid oxidation and inhibits energy-consuming pathways such as fatty acid synthesis
Agm is more consistently up-regulated after brain injury, whereupon it seems to serve a protective function by dampening NMDA-NO excitotoxicity, suggesting possible roles of co-transmission role with Glu, but also potentially NOS inhibition (Kuo et al., 2007
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[4] Niacinamide (vitamin B) forms the coenzymes NAD and NADP, which are involved in hundreds of oxidation-reduction reactions in cells
2011 Oct;339(1):35-44
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