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The somatomedin hypothesis which was first proposed in the early 1970s, provided a model for IGF1 actions on the skeleton (see Figure 2 ) elaborated in igf-1 global knockouts, as will be discussed in more detail in a later section, show infertility and profound growth retardation at birth, phenotypes that are not observed in GH- or GH receptor-null animals
Moreover, poor responsiveness, pharmacological tolerance, risk of poisoning, high risk of misuse and improper use (e.g
Concise review: mechanisms of quiescent hair follicle stem cell regulation
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perhaps a slight testosterone dip or cholesterol change, rather than complete shutdown of hormones
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