Exercise as a Secondary Metabolic Stressor Adipose tissue dysfunction in obesity promotes the release of small extracellular vesicles enriched with oxidatively stressed mitochondrial components [66, 91], reflecting unresolved mitochondrial strain
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After -oxidation, butyrate will eventually produce two molecules of acetyl-CoA [21], whose concentration directly affects the level of protein acetylation
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Endocrinology (1985) 117:117983
