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Growth is supported by Germany's early adoption of cardiovascular outcome data into treatment guidelines and the country's structured reimbursement pathways for innovative therapies [1]
The clinical trade-off: higher concentrations provide stronger methylation pathway support but require less frequent dosing
Taken together, these in vivo data from multiple CRC models support our conclusion that inhibition of mitochondrial metabolism by metformin treatment can enhance the response of 5FU treatment by increasing tumour cell death and impeding tumour growth
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