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Further research using BNC1 / mice demonstrated that BNC1 defects promote intracellular iron accumulation and induce oocyte ferroptosis by activating the Merlin (NF2)/Hippo/YAP/TFRC/ACSL4 pathway (Wang F

J Membr Biol 89:18 Lu SC, Ge JL, Kuhlenkamp J, Kaplowitz N (1992) Insulin and glucocorticoid dependence of hepatic gamma-glutamylcysteine synthetase and glutathione synthesis in the rat

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Mccall also shares her personal connection to the topic through her daughter Rosie, highlighting the potential impact of these approaches for families facing health challenges