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Zorov DB, Juhaszova M, Sollott SJ

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Written By: Jeffrey Atlas, PA-C, Health Content Writer Medically Reviewed By: Dr

doi: 10.3390/cancers12061607 208 LeaoICGanesanPArmstrongTDJaffeeEM

Tubular epithelial fibroblast transdifferentiation (TEMT) has been proposed as a viable therapeutic option to decrease RF since it stimulates the expression of several fibro-cytokines, which represent a critical mechanism in the development of RF ( SalB mitigates TEMT and has renal protective effects by down-regulating the expression of FGF2/TGF-1/-SMA via the HPSE/Syndecan-l axis ( 2.4.3 PDGF PDGF is a profibrotic mediator that induces fibroblast activation and proliferation and promotes ECM contraction ( SalB in reducing renal inflammation and CTGF production, improving renal function, and inhibiting the progression of RF through the PDGF-C/PDGFR pathway has been well-documented (Yao et al., 2022)

This reaction breaks the disulfide bonds, converting them into two thiol groups, and facilitates targeted drug release where it is needed most