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This results in increased proteasomal degradation of oxidatively damaged proteins as well as p62, thereby facilitating ATG-lysosomal degradation of proteins and dysfunctional organelles, such as the mitochondria (Levonen et al., 2014)
Giornale Italiano di Dermatologia e Venereologia
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AJR Am J Roentgenol
This imbalance disrupts the follicular microenvironment and diminishes long-term oocyte developmental potential (Fig
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