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doi:10.1080/09546634.2018.1444728
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This experimental models ability to allow systematic variation of substrate end structure, initiation of cellular repair of these substrates with high synchrony (reactions start within minutes), and the ability to track each strands repair product in parallel was essential
It's helpful to see how it stacks up against other well-known compounds
This compound treatment (3.12-100 g/mL, 24-48 h) inhibited cell viability in a dose- and time-dependent manner, induced apoptosis, and caused G0/G1 phase cell cycle arrest in A549 cells [2]
He received his medical degree from the Stanford University School of Medicine and trained at the Johns Hopkins Hospital in general surgery
