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Subsequently, changes in mitochondrial membrane potential of liver cells after METH and QR treatment were assessed using Rhodamine 123 and mitochondrial red fluorescence probes, respectively (Figures 4C, D)

The G6PD deficiency is produced by different genetic mechanisms such as deletions, precise mutations and substitutions that affect the transcription, process or primary enzyme structure, which functionally results in a decrease of the enzymatic activity or loss of affinity by the substrate