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While traditional models often address hyperandrogenism and IR as primary, parallel drivers of PCOS pathology, they frequently fail to account for the rising evidence of systemic low-grade inflammation and immune dysregulation in the syndrome, particularly the pervasive role of the gut-metabolic axis [2, 16, 17]

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While the physiological pathways MOTS-c influences, such as the AMPK pathway, are highly conserved across species, the pharmacokinetics and ultimate clinical efficacy can differ significantly between mice and humans

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doi: 10.1371/journal.pone.0208906 126 LeeuwendaalNKHayesJJStantonCOToolePWBeresfordTP