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When this happens, bile can no longer flow normally, potentially leading to inflammation or infection of the gallbladder, a condition called acute cholecystitis

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This relatively small GPCR family is characterized by a long extracellular ligand-binding domain stabilized by three disulfide bridges formed by six highly conserved cysteine residues, seven -helical transmembrane domains, universal for all GPCRs and an intracellular C-terminal domain, responsible mostly for receptor internalization due to the presence of multiple phosphorylation sites [22, 23]

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In the last years, many trials failed to find a unique treatment for all HFpEF phenotypes, apart from sodium-glucose cotransporter 2 inhibitors

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