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Van Calcar SC et al
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Therefore, therapeutic strategies that target the distinct mitochondrial metabolic features of CSCs and DTPs, such as disrupting the AMPKOXPHOS axis, inhibiting mitophagy, or decoupling metabolic signaling from DNA repair, represent a promising approach to overcoming chemoresistance in CRC
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In conclusion, these findings suggest that targeting IGFs is important for inhibiting cancer proliferation and aggressiveness
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