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More interestingly, a few metabolites (e.g., plasma histidine, erythroid carnitine derivatives) were reported to exclusively differ in PO+ children, whereas similar levels were detected in control and PO subjects, thus suggesting that PO might play an important role in modulating these metabolic programming mechanisms
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However, in 2001, a handful of groundbreaking research discoveries confirmed that the Ca 2+ ionophore ionomycin could trigger an apoptosis-like cell death in erythrocytes associated with the appearance of apoptotic signs, including cell shrinkage, membrane vesiculation, or PS exposure [22, 23]
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[PMID: 31379746] PubMed record related to this compound for laboratory literature review
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