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Tan IIA induced activation of the GSH system to attenuate cardiac oxidative stress and restrain the increase in serum myocardial enzymes, thus relieving the extent of myocardial damage [154, 155]

Cohort composition changed materially during the window

Zeng X, Qiu D, Hu R, Zhang M

(124) discovered that iron-catalyzed apoptosis might be induced by the degradation of manganese-silica nanoparticles themselves (through breakage of Mn-Obond)