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doi: 10.1038/nrendo.2012.140 115 DruckerDJBuseJBTaylorKKendallDMTrautmannMZhuangDet al
Proponents of this view suggest that neuronal energy demands during activation are met directly by glucose or other mechanisms, minimizing the quantitative importance of an intercellular lactate shuttle (Dienel, 2012, 2017)
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All current evidence comes from preclinical animal and cellular studies, meaning its efficacy and safety in people remain unproven
we also determined whether the possible underlying mechanisms of its effects were associated with Nrf2-regulated anti-oxidative, mTOR-mediated anti-apoptotic, and/or autophagy systems
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