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A phase 1/2 trial of HQK-1001, an oral fetal globin inducer, in sickle cell disease
The corrected effect size for publication bias showed significant changes after trim and fill analysis (WMD = 1385 mm
The PDS develops from large and relatively slow excitatory postsynaptic potentials mediated by AMPA and NMDA glutamate receptors activated by aberrant and large glutamate release within the foci
The specific mechanisms remain under investigation, but the finding adds another dimension to DSIP's systemic regulatory effects
Recent research by Sim et al
In normoxia, hydroxylation of HIF- by PHDs enables its interaction with pVHL (von Hippel-Lindau protein) and targets subunits for ubiquitination and proteasomal degradation [140]
