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Ethanol metabolism, CYP2E1 induction, compromised antioxidant defenses, mitochondrial injury, activation of Kupffer and stellate cells, hypoxia, and iron overload can all contribute to the alcohol-induced oxidative environment
Shen et al., 2016)
121(5) , e2305770121.10.1073/pnas.2305770121 50 LiangY
They also interact with receptors in the brain to increase the feeling of being sated
Thats a good thing the less sugar we have to burn, the less insulin we have to produce and that means protection against prediabetes, metabolic syndrome and type 2 diabetes
The FDA does not explicitly require a single fixed-dose maintenance strategy
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