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Rather than simply increasing or decreasing NO production, studies indicate that the compound modulates NO activity contextually, supporting vasodilation and blood flow where healing requires it while counteracting NO-mediated tissue damage in inflammatory contexts (3)

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Axonal transport can be disrupted through various mechanisms, including damage to motor proteins and cytoplasmic dynein, microtubules, cargo, and mitochondria [42, 43]

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USA 110 , 88828887 (2013)