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Remarkably, the impact of parental germ cells on TL remains significant even despite the telomere reprogramming throughout embryonic development, initially by the recombination-based mechanisms and then through the activity of telomerase at the blastocyst stage and later (Kalmbach et al., 2014, see also Figure 2 for schematic illustration)

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Each targets different aspects of the tissue repair process, which is why they are sometimes discussed together in regenerative medicine contexts

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