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Dihexa acts primarily through hepatocyte growth factor (HGF) signaling by potentiating HGF/c-Met receptor interaction

Published MT-II literature often frames the compound through cyclic peptide structure, melanocortin receptor activity, and analog design [6] [13]

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Oxidative stress, induced by ROS, triggered the translocation of Fas into lipid rafts, the formation of a Fas-associated signaling complex, and the subsequent activation of caspases-8 and -3

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