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Notably, this protective effect occurred through mechanisms independent of changes in muscle oxidative metabolism or fatigue resistance, distinguishing IGF-1 LR3's mechanism from native IGF-1

Inhibition of IL-1 secretion by GSTO1 inhibitors also has exciting implications on tumour progression and tumour-immune cell crosstalk in the microenvironment

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Therefore both GST and efflux pumps overexpression may confer high levels of resistance to the cytotoxic action of several antineoplastic drugs

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