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The formation of desacyl-ghrelin is the consequence of alternative splicing due to an intron that reside between the glutamines at positions 13 and 14 (Q13 and Q14) of the preproghrelin sequence
This crosstalk amplifies neuroinflammatory signaling and alters neurovascular integrity, contributing to blood-brain barrier disruption, glial priming, and neuronal injury 11-12
DaoKShechtmanSWeber-SchoendorferCDiav-CitrinOMuradRBerlinMet al
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