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For in-vitro research planning only
The study was designed as proof of concept for NNMT as a drug-development target
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Cellular signaling mechanisms
The postulated mechanism for causing such attacks is through stimulation of the porphyrin precursor, delta-aminolevulinic acid (ALA) Laboratory tests: Diclofenac prolongs platelet aggregation without affecting the bleeding time, plasma fibrinogen and factors V and VII to XII
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