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GSH dysregulation might cause global immune cell autophagy decline with increased generation of proinflammatory cytokines in aging, further provoked by mitochondrial ROS signaling (293)

The treated fat does not return

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Taken together, these outcomes are consistent with a scenario in which NF-Bdependent inflammatory activity is dampened, Nrf2-mediated antioxidant defenses are strengthened, and TGF-/Smad signaling is reduced

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This DMF concentration didnt reduce cell viability under non-BSO pretreatment condition and reduced cell viability less than 5% under BSO pretreatment condition (Supplementary Figure S2)