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, & Head, K
In hyperhomocysteinemia-induced myocardial injury, HS mitigates pathological progression through a four-pronged mechanism: (1) restoring CSE activity to lower plasma homocysteine levels, (2) reducing oxidative stress by decreasing lipid peroxidation product levels, (3) enhancing ROS scavenging through restored mitochondrial respiratory enzyme activity, and (4) inhibiting expression of ER stress markers (Xia et al., 2023)
Glutathione supplements, especially when taken orally at average doses, are well tolerated by most people
Taken together, these data demonstrate that mitochondrial function is intrinsically linked to 5FU sensitivity, and that its inhibition may provide an opportunity to increase 5FU efficacy
Using the daily protocol for the weekly version (CJC-1295 w/ DAC) or vice-versa is a serious error
